By I. Klatzo (auth.), K. Schürmann, M. Brock, H.-J. Reulen, D. Voth (eds.)
This quantity comprises the papers awarded on the twenty fourth Annual assembly of the Deutsche Gesellschaft fur Neurochirurgie, held in Mainz, Western Germany, on April 30 - may possibly three, 1973. planned selection used to be made from an important nonetheless hotly debated topics which, for a while, have intended a resource of continuing fear, and nights with no sleep to each neurosurgeon. simply so long as our certain box exists, there were the issues of ways to regulate mind edema and ·of the best way to decrease lethality and the secondary lesions in surgical procedure of cerebello-pontine attitude tumors. about the first topic, new pathological, pathophysiological and chemical elements, the mechanisms of mind edema formation and determination are awarded within the desire for higher figuring out. moreover, the connection among mind edema, intracranial strain, cerebral blood stream and metabolism are mentioned. eventually, the therapeutical effects in addition to the result of experimental and medical paintings are awarded, and a comparability of results among diversified tools (hypertonic suggestions, diuretics, steroids, managed hyperventilation, hyperbaric oxygen) is given. in regards to the moment major topic, any very important contributions to the early prognosis of cerebello-pontine attitude tumors were integrated. however, it's of maximum curiosity for the neurosurgeon to understand which procedure he's to favor for the several phases of tumor measurement and to be acquainted with the trans labyrinthine procedure or the posterior craniotomy, in addition to with the significance of using the microscope in neurosurgery, the upkeep of the facial nerve and, on occasion, its repair.
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Extra resources for Brain Edema / Cerebello Pontine Angle Tumors: Pathophysiology and Therapy / Diagnosis and Surgery
12 hours after cerebral ischemia. Swelling of the capillary endothelium. x 4, 500 Fig. 3. Cat. six hours after cerebral ischemia. Hemorrhages in the midbrain and in medial parts of the occipital lobe Blood Brain Barrier and Scintigraphy L. PENNING and D. FRONT A scintigraphic study of brain contusion confronted us again with the problem of the blood-brain barrier. It is generally accepted that damage of the blood-brain barrier is a cause of scintigraphic visualization of a brain lesion. However, the problem in brain tumors is quite different from that in brain contusion.
Though in our experiments obstruction of capillaries was confined to such nonperfused areas we think that they were the result rather than the cause of the "no-reflow" phenomenon. The distribution of lesions makes it probable that first venous compression by brain edema caused a regional stop of the circulation, which was followed by lesions of the microvasculature. Severe brain edema after cerebral ischemia or anoxia develops in two phases. 21 First intracranial hypertension is confined to the supratentorial space.
Path. Bact. 90, 533-541 (1965) 16. STOECKEL, H. and S. HOYER: Hirndurchb1utung und Hirnstoffwechse1 nach schwerert HirntraUInen. An~sth. und Wiederbel. 6, 431 (1971) 17. SCHWEIZER, O. and W. S. HOWLAND: Prognostic Significance of High Lactate Levels. Ana1g. 47, 383-388 (1968) 18. VITEK, V. A. COVLEY: Blood lactate in the prognosis of various forIns of shock. Surg. 173, 308-313 (1971) 19. WElL, M. H. and A. A. AFIFI: ExperiInenta1 and clinical studies on lactate and pyruvate as indicators of the severity of acute circulatory failure (shock), Circulation 41, 989-1001 (1970) 20.