By Brian C. J. Moore
Because the first version used to be released in 1998, significant advances were made within the fields of pitch conception and speech notion. moreover, there were significant adjustments within the method that listening to aids paintings, and the positive aspects they give. This ebook will offer an figuring out of the alterations in conception that ensue while somebody has cochlear listening to loss so the reader is familiar with not just what does occur, yet why it occurs. It interrelates physiological and perceptual information and offers either this and uncomplicated thoughts in an built-in demeanour. The objective is to exhibit an realizing of the perceptual alterations linked to cochlear listening to loss, of the problems confronted by means of the hearing-impaired individual, and the constraints of present listening to aids.
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Additional resources for Cochlear Hearing Loss: Physiological, Psychological and Technical Issues (Wiley Series in Human Communication Science)
Similarly, if there are no functioning IHCs at an apical region of the 44 COCHLEAR HEARING LOSS cochlea, a low-frequency sound may be detected via neurones that are tuned to higher frequencies. Because of this possibility, the ‘true’ hearing loss at a given frequency may be greater than suggested by the audiometric threshold at that frequency. A region of the BM over which there are no functioning IHCs is referred to as a dead region (Moore and Glasberg, 1997). ’ Often the extent of a dead region is described in terms of the CFs of the functioning IHCs or neurones at the edge of the dead region (Moore, 2001).
Also, for high CFs, the frequency at the tip (the lowest point on the curve) may decrease slightly with increases in the predetermined firing rate. This reflects the change in BM tuning with level described earlier. 14 shows schematically how the rate of discharge for three auditory nerve fibres changes as a function of stimulus level. The curves are called rate-versus-level functions. In each case, the stimulus was a sinusoid at the CF of the neurone. Consider first the curve labelled (a). This curve is typical of what is observed for neurones with high spontaneous firing rates.
In such cases, vibration at those places is not detected by the neurones directly innervating them. Say, for example, the IHCs at the basal end of the cochlea are non-functioning. Neurones innervating the basal end, which would normally have high characteristic frequencies (CFs), will not respond. However, if a high-frequency sinusoid is presented, it may be detected if it produces sufficient BM vibration at a more apical region; this corresponds to downward spread of excitation. In other words, a high-frequency sound may be detected via neurones that are tuned to lower frequencies.