By Zhou Xuedong
-- Covers many points of dental caries
-- offers precious images to illustrate number of medical manifestations and complications
-- bargains with vital present concerns in an intensive yet undemanding and easy-to-read way
l-illustrated and complete consultant to the etiology, medical manifestations, prognosis, scientific administration and prevention of dental caries. present not easy difficulties within the box are analyzed and the most recent learn findings, offered. After an introductory bankruptcy on enamel improvement, the relationships of biofilm and saliva to dental caries and the importance of the stability among demineralization and remineralization for the improvement of carious lesions are mentioned. next chapters handle the cutting-edge in prognosis and remedy, the results of disorder burden for prevention and the organization among systemic ailments and dental caries. Dental Caries: rules and administration is meant for dental university scholars, training dentists and researchers in dentistry.
-- Oral and Maxillofacial surgical procedure
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Additional info for Dental Caries: Principles and Management
1996;7(2):104–33. 33. Forssell-Ahlberg K, Brannström M, Edwall L. The diameter and number of dentinal tubules in rat, cat, dog and monkey: a comparative scanning electron microscopic study. Acta Odontol. 1975;33:243–50. 34. Mjör IA, Pindborg JJ. Histology of the human tooth. Copenhagen: Munksgaard; 1973. 35. Bonucci E. Matrix vesicles: their role in calcification. Dentin Dentinogen. 1984;1:135–54. 36. Stanley H, Pereira J, Spiegel E, Broom C, Schultz M. The detection and prevalence of reactive and physiologic sclerotic dentin, reparative dentin and dead tracts beneath various types of dental lesions according to tooth surface and age.
These fascinating observations at the birth of microbiology had already signaled the complexity of the oral microbial community . microorganisms can convert carbohydrate into acid and eventually result in the demineralization of teeth and development of dental caries in susceptible hosts. Miller’s chemoparasitic theory, together with the description of “gelatinous microbic plaques”, which are now commonly known as “dental plaque,” by Black and Williams [16, 219], has laid the foundation for our modern knowledge of dental caries etiology.
When bone resorbed on the coronal side, bone formed on the basal side of the tooth. It is thought that the dental follicle, which is a loose connective tissue sac surrounding the enamel organ of each tooth, plays a critical role in regulating osteoclasts and osteoblasts during this process. In 1980s, it was firstly shown that the removal of the dental follicle from developing premolars of dogs prevented tooth eruption. Meanwhile, if the tooth germ was removed and replace with another artificial tooth, which the dental follicle was kept intact in situ, the artificial tooth still erupted on schedule.